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January 10, 2023

What a Collision Does, and Why Some Pain Stays

by - Dr. Gurpreet Singh Padda, MD, MBA, MHP

A collision is an event that lasts about a fifth of a second. The injury it causes is a process that runs for months, and almost everything that decides how that process ends happens after everyone has gone home and the car has been towed.

This page is the orientation: where the force actually goes, why two people in the same vehicle walk away with different injuries, and what separates the collisions that resolve from the ones that turn into a pain problem.

Where the force actually goes

Injury in a crash arrives by two routes. Blunt impact is the obvious one: you strike the wheel, the pillar, the road. Inertial loading is the one that fills pain clinics — nothing strikes you at all, and the mass of your own head and trunk strains the tissue holding it.

The neck is where that shows up most reliably. During impact the cervical spine passes through a transient S-shaped curvature, the lower segments extending while the upper segments are still flexed, so the neck briefly holds a shape it never adopts in voluntary movement. That curvature and the tissue strains that accompany it have been documented in rear, frontal and side impacts. It is finished before a protective muscle response can be organized.

What gets strained is joint capsule, ligament and small stabilizing muscle. None of that appears on the imaging ordered in an emergency department, because that imaging exists to exclude fracture and instability — a different question, correctly asked and correctly answered.

Why two people in the same car do not get the same injury

Crash-injury research is explicit about something patients are rarely told: there is no injury tolerance that applies to everyone in a vehicle. What happens to a given occupant depends on three separate stacks of variables.

  • Who you are — age, height, weight, sex, bone mineral density, and whatever musculoskeletal and medical conditions you brought into the seat with you.
  • What was true a second before impact — whether you saw it coming, how you were sitting, where the seatbelt sat, where the head restraint sat relative to the back of your skull, and the vehicle itself.
  • The collision — its direction, the vehicle’s dynamics, which safety systems deployed, and how your body moved in response.

This is why the passenger is fine and the driver is not, and why an estimate written about sheet metal is not a statement about a cervical spine. It is also why the intake questions about where you were looking and whether you were braced are clinical data rather than conversation.

The first six weeks are not a waiting period

The instinct after a crash is to wait and see. The evidence says the early period carries the information.

The Bone and Joint Decade Task Force reviewed the literature on whiplash-associated disorders and found that greater initial pain, more symptoms at the outset, and greater initial disability predicted slower recovery, while few characteristics of the collision itself were prognostic at all. A later systematic review agreed: early pain-related disability and injury grade track with outcome, while neck range of motion, joint position sense and muscle strength do not reliably predict anything.

So the severity of what you feel in week one is worth measuring properly, and the collision details everyone argues about are worth less than they are given credit for. Waiting three weeks to be seen destroys the only measurement that had predictive value.

When it does not resolve, it is usually a structure you can name

Persistent pain after a collision is not a mystery diagnosis. It is a short list of structures, and each can be tested.

In chronic neck pain after whiplash, the cervical facet joints have been identified as the source in roughly 54% to 60% of patients. The way that is established is a medial branch block — anesthetize the small nerve that serves the joint and observe whether the pain leaves on the anesthetic’s schedule and returns on it. A single block is wrong about 40% of the time, which is why controlled blocks are the standard before anything durable is done. How a medial branch block is performed and read, and what follows a positive one.

The same discipline applies elsewhere. Arm pain after a crash may be a nerve root rather than a neck joint (cervical radiculopathy). A headache starting at the base of the skull is often cervicogenic rather than migraine (headache that starts in the neck). Numb fingers may be the wrist or the neck, and the two are told apart by testing (hand numbness).

The part imaging keeps missing

Beyond the joints, the deep stabilizing muscles of the neck do not simply resume work. Reduced cervical mobility, disturbed position sense and altered muscle activity are measurable soon after a whiplash injury and persist in those with moderate to severe symptoms — while being poor predictors of who will end up in that group.

Read that carefully, because it cuts both ways. The dysfunction is real and worth rehabilitating. It is also not a test that tells you your future, and any clinic selling it as one is overselling.

What actually decides the year after a collision

Two things that rarely appear in an accident report.

Behavior. The same Task Force review found that passive coping, depressed mood and fear of movement predicted slower and less complete recovery. That is not a suggestion the pain is invented — the tissue injury is documented in the same literature. It means guarding and avoidance are treatable drivers, and treating them is part of the medicine. Acceptance and Commitment Therapy is delivered here in-house for exactly this reason.

The biological terrain. Strained tissue heals in whatever metabolic state it finds. Systemic inflammation, insulin resistance and wrecked sleep slow the repair of a joint capsule the same way they slow everything else, and sleep is the input that collapses first after a crash and gets treated last. Why metabolic inflammation governs how pain behaves.

What to do, in order

  1. Be evaluated within days, not weeks, even if you feel able to work.
  2. Give the mechanism, not just the symptom: direction of impact, seating position, where you were looking, whether you were braced, head restraint position, airbag deployment, whether your head struck anything.
  3. Report the whole picture — sleep, concentration, mood, light sensitivity, what you have stopped doing. Those carry prognostic weight and get edited out because they sound like complaining.
  4. Keep moving within tolerance. Protective immobilization past the first days feeds the fear-avoidance loop the evidence identifies as a driver of poor recovery.
  5. If pain is still present at six to eight weeks, stop treating it generically and get the generator identified. Neck pain months after a car accident is a solvable problem when it is named.

Care after a collision at this practice follows that sequence: examination first, imaging read against the examination rather than in isolation, electrodiagnostic testing when the question is nerve, diagnostic blocks when the question is which joint, and rehabilitation and metabolic work running alongside rather than after. What that looks like in practice.

Frequently asked questions

Can a collision injure me if the car was barely damaged?

Yes. Published crash-injury work states plainly that no single injury tolerance applies to all occupants — outcome depends on your age, sex, bone density and pre-existing conditions, on how you were seated and whether you saw it coming, and on the crash direction and safety systems. Vehicle damage and occupant injury are different measurements. Learn more: whiplash evaluation and treatment.

My neck X-ray and CT were normal. Why does it still hurt?

Because those studies are ordered to exclude fracture and instability, and they answer that question well. Joint capsule strain, ligament injury and deep stabilizing muscle dysfunction — the tissues loaded during the transient S-shaped curvature of a crash — are not what they show. Learn more: neck pain.

How long should pain after a crash take to settle?

Most improvement happens in the first weeks. The evidence on whiplash-associated disorders finds greater initial pain, more symptoms and greater initial disability predict slower recovery, so pain that is still limiting you at six to eight weeks is a reason to identify the pain generator rather than to keep waiting. Learn more: neck pain months after a car accident.

How do you find out which structure is causing the pain?

By blocking it. Cervical facet joints are the source in roughly 54% to 60% of chronic post-whiplash neck pain, and a medial branch block tests that directly — the pain should leave and return on the anesthetic’s schedule. Single blocks are wrong about 40% of the time, so controlled blocks are used before anything durable. Learn more: facet joint pain and the medial branch block.

Does treating the psychological side mean my pain is not physical?

No. The same body of evidence documents the tissue injury and identifies fear of movement, passive coping and depressed mood as predictors of slower recovery. They are treatment targets sitting alongside the physical ones, not a verdict about whether you were hurt. Learn more: Acceptance and Commitment Therapy for chronic pain.

I was hurt at work rather than on the road. Is any of this different?

The biomechanics and the diagnostic sequence are the same; the paperwork is not, and which state the injury happened in changes it further. The clinical approach — identify the generator, treat it, document function over time — does not change. Learn more: work injury pain management.

Sources

  1. National Highway Traffic Safety Administration. “Summary of Motor Vehicle Traffic Crashes: 2023 Data.” DOT HS 813 762. October 2025. crashstats.nhtsa.dot.gov
  2. Ivancic PC. “Mechanisms and mitigation of head and spinal injuries due to motor vehicle crashes.” Journal of Orthopaedic & Sports Physical Therapy. 2016;46(10):826–833. doi:10.2519/jospt.2016.6716
  3. Carroll LJ, Holm LW, Hogg-Johnson S, et al. “Course and prognostic factors for neck pain in whiplash-associated disorders (WAD).” Spine. 2008;33(4 Suppl):S83–S92. doi:10.1097/BRS.0b013e3181643eb8
  4. Alalawi A, Mazaheri M, Gallina A, et al. “Are measures of physical function of the neck region associated with poor prognosis following a whiplash trauma? A systematic review.” The Clinical Journal of Pain. 2022;38(3):208–221. doi:10.1097/AJP.0000000000001015
  5. Manchikanti L, Singh V, Rivera J, Pampati V. “Prevalence of cervical facet joint pain in chronic neck pain.” Pain Physician. 2002;5(3):243–249. PMID 16902649
  6. Daenen L, Nijs J, Raadsen B, et al. “Cervical motor dysfunction and its predictive value for long-term recovery in patients with acute whiplash-associated disorders: a systematic review.” Journal of Rehabilitation Medicine. 2013;45(2):113–122. doi:10.2340/16501977-1091

Dr. Gurpreet Singh Padda, MD, MBA, MHP

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